你给的链接，
https://www.masslive.com/news/2022/10/b ... versy.html
内容copy如下
Boston University is defending its research after a report studying the COVID-19 omicron variant’s lower severe infection rate was met with criticism among news outlets and one of its financial backers.
BU’s National Emerging Infectious Diseases Laboratories removed an omicron variant spike protein and attached it to a virus of the original COVID strain. This fused virus, as Boston.com reported, was then found to be more lethal than the naturally occurring omicron variant when exposed to lab mice, in contrast to 100% with the original strain.
“The Omicron S-bearing virus robustly escapes vaccine-induced humoral immunity, mainly due to mutations in the receptor-binding motif... while Omicron causes mild, non-fatal infection, the Omicron S-carrying virus inflicts severe disease with a mortality rate of 80%,” the report says.
The findings of the report, which has not yet been peer-reviewed, concluded that mutations of the omicron spike allow the variant to evade any developed immunity and are not the cause of omicron’s lesser severity compared with other variants.
Some headlines have called the research “playing with fire” or alleged that the university has claimed outright that they have developed a lethal COVID strain. Boston University first clarified in a statement that the research was not gain-of-function research, and therefore did not make a more severe version of the omicron variant, instead making “the virus replicate less dangerous.”
Read more: Mass. gets $25 million for research center to fend off future pandemic
“Secondly, the research was reviewed and approved by the Institutional Biosafety Committee (IBC), which consists of scientists as well as local community members,” the statement continued. “The Boston Public Health Commission also approved the research. Furthermore, this research mirrors and reinforces the findings of other, similar research performed by other organizations, including the FDA. Ultimately, this research will provide a public benefit by leading to better, targeted therapeutic interventions to help fight against future pandemics.”
“They’ve sensationalized the message, they misrepresent the study and its goals in its entirety,” said Ronald Corley, BU Chobanian and Avedisian School of Medicine chair of microbiology, when speaking with Boston University research’s news website The Brink.
The research’s existence has also allegedly caused a rift between BU researchers and the National Institute of Allergy and Infectious Diseases. NIAID helped fund their work, but Director of NIAID’s Division of Microbiology and Infectious Diseases Emily Erbelding told STAT that there was no specific mention of their exact work in the grant application.
“I think we’re going to have conversations over upcoming days,” Erbelding said to STAT.
Researchers said the study was conducted in the lab’s biosafety-level 3 facilities, entering and exiting their workspace through several interlocked doors, according to The Brink. The space is completely sealed and the lab is fitted with proper, sophisticated filtration and decontamination technology.
“We take our safety and security of how we handle pathogens seriously, and the virus does not leave the laboratory in which it’s being studied,” Corley said to The Brink. “Our whole goal is for the public’s health. And this study was part of that, finding what part of the virus is responsible for causing severe disease. If we can understand that, we can then develop the tools that we need to develop better therapeutics.”
google翻译
在一份研究 COVID-19 omicron 变体的较低严重感染率的报告遭到新闻媒体及其一位财务支持者的批评后，波士顿大学正在为其研究辩护。
BU 的国家新兴传染病实验室移除了一种 omicron 变异刺突蛋白，并将其附着在原始 COVID 毒株的病毒上。正如 Boston.com 报道的那样，这种融合病毒随后被发现在暴露于实验室小鼠时比天然存在的 omicron 变体更致命，而原始毒株则为 100%。
“携带 Omicron S 的病毒强烈逃避疫苗诱导的体液免疫，主要是由于受体结合基序的突变……而 Omicron 会导致轻度、非致命的感染，而携带 Omicron S 的病毒会导致严重的疾病并致死80%，”报告说。
该报告的结果尚未经过同行评审，其结论是，与其他变体相比，omicron 尖峰的突变允许该变体逃避任何已形成的免疫，并且不是 omicron 严重程度较低的原因。
一些头条新闻称这项研究是“玩火”，或声称该大学直接声称他们已经开发出一种致命的 COVID 毒株。波士顿大学首先在一份声明中澄清说，这项研究不是功能获得性研究，因此没有制造出更严重的 omicron 变体版本，而是降低了“病毒复制的危险性”。
阅读更多：马萨诸塞州获得 2500 万美元用于研究中心以抵御未来的流行病
“其次，该研究得到了机构生物安全委员会（IBC）的审查和批准，该委员会由科学家和当地社区成员组成，”声明继续说道。 “波士顿公共卫生委员会也批准了这项研究。此外，这项研究反映并强化了其他组织（包括 FDA）进行的其他类似研究的结果。最终，这项研究将通过带来更好的、有针对性的治疗干预措施来帮助对抗未来的流行病，从而提供公共利益。”
“他们夸大了这个信息，他们歪曲了这项研究及其整体目标，” BU Chobanian 和 Avedisian 医学院微生物学主席 Ronald Corley 在与波士顿大学研究新闻网站 The Brink 交谈时说。
据称，这项研究的存在还导致了 BU 研究人员与国家过敏和传染病研究所之间的裂痕。 NIAID 帮助资助了他们的工作，但 NIAID 微生物学和传染病部主任 Emily Erbelding 告诉 STAT，在拨款申请中没有具体提及他们的确切工作。
“我认为我们将在未来几天进行对话，”Erbelding 对 STAT 说。
据 The Brink 称，研究人员表示，这项研究是在实验室的 3 级生物安全设施中进行的，通过几个互锁的门进出他们的工作区。空间完全密封，实验室配备了适当、先进的过滤和净化技术。
“我们认真对待我们如何处理病原体的安全和保障，病毒不会离开正在研究它的实验室，”科利对 The Brink 说。 “我们的全部目标是为了公众的健康。这项研究是其中的一部分，它发现了病毒的哪一部分是导致严重疾病的原因。如果我们能理解这一点，我们就可以开发出开发更好疗法所需的工具。”
哪里说了武汉改造舟山蝙蝠了？
1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
版主： verdelite， TheMatrix
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
不是写的清清楚楚，removed an omicron variant spike protein and attached it to a virus of the original COVID strain original COVID strain。
original covid strain 不就是武汉病毒吗。
original covid strain 不就是武汉病毒吗。
上次由 ghobuster 在 2022年 10月 18日 19:13 修改。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
LOL，原来是屁股坐歪了的臆测。
original COVID strain，就算是武汉毒株，能推出 武汉改造了舟山蝙蝠吗?
☭主体思想の光辉万万年！
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
中共和WHO打杂客，咬死了不可能造出人工病毒，这不就轻轻松松造了一个出来了吗。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
这货的帖从来不点，要在老版大灌早被叔黑了
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
瑞典早就用酵母菌搬运复刻了武汉毒株。
土工只是说自己没能力造，没说美帝没能力造。
你就是故意混淆概念。
☭主体思想の光辉万万年！
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
那是因为你们根本就没办法解释。你回我了，反而更多人看到。还不如掩耳盗铃了。
不如，你也让楼主回去培训一下。
不如，你也让楼主回去培训一下。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
那为什么早先一群五毛说，这病毒无法人工制造？
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
1450 造谣一时爽，ghobuster 全家火葬场
laojin 写了： 2022年 10月 18日 19:08 你给的链接，
https://www.masslive.com/news/2022/10/b ... versy.html
内容copy如下
Boston University is defending its research after a report studying the COVID-19 omicron variant’s lower severe infection rate was met with criticism among news outlets and one of its financial backers.
BU’s National Emerging Infectious Diseases Laboratories removed an omicron variant spike protein and attached it to a virus of the original COVID strain. This fused virus, as Boston.com reported, was then found to be more lethal than the naturally occurring omicron variant when exposed to lab mice, in contrast to 100% with the original strain.
“The Omicron S-bearing virus robustly escapes vaccine-induced humoral immunity, mainly due to mutations in the receptor-binding motif... while Omicron causes mild, non-fatal infection, the Omicron S-carrying virus inflicts severe disease with a mortality rate of 80%,” the report says.
The findings of the report, which has not yet been peer-reviewed, concluded that mutations of the omicron spike allow the variant to evade any developed immunity and are not the cause of omicron’s lesser severity compared with other variants.
Some headlines have called the research “playing with fire” or alleged that the university has claimed outright that they have developed a lethal COVID strain. Boston University first clarified in a statement that the research was not gain-of-function research, and therefore did not make a more severe version of the omicron variant, instead making “the virus replicate less dangerous.”
Read more: Mass. gets $25 million for research center to fend off future pandemic
“Secondly, the research was reviewed and approved by the Institutional Biosafety Committee (IBC), which consists of scientists as well as local community members,” the statement continued. “The Boston Public Health Commission also approved the research. Furthermore, this research mirrors and reinforces the findings of other, similar research performed by other organizations, including the FDA. Ultimately, this research will provide a public benefit by leading to better, targeted therapeutic interventions to help fight against future pandemics.”
“They’ve sensationalized the message, they misrepresent the study and its goals in its entirety,” said Ronald Corley, BU Chobanian and Avedisian School of Medicine chair of microbiology, when speaking with Boston University research’s news website The Brink.
The research’s existence has also allegedly caused a rift between BU researchers and the National Institute of Allergy and Infectious Diseases. NIAID helped fund their work, but Director of NIAID’s Division of Microbiology and Infectious Diseases Emily Erbelding told STAT that there was no specific mention of their exact work in the grant application.
“I think we’re going to have conversations over upcoming days,” Erbelding said to STAT.
Researchers said the study was conducted in the lab’s biosafety-level 3 facilities, entering and exiting their workspace through several interlocked doors, according to The Brink. The space is completely sealed and the lab is fitted with proper, sophisticated filtration and decontamination technology.
“We take our safety and security of how we handle pathogens seriously, and the virus does not leave the laboratory in which it’s being studied,” Corley said to The Brink. “Our whole goal is for the public’s health. And this study was part of that, finding what part of the virus is responsible for causing severe disease. If we can understand that, we can then develop the tools that we need to develop better therapeutics.”
google翻译
在一份研究 COVID-19 omicron 变体的较低严重感染率的报告遭到新闻媒体及其一位财务支持者的批评后，波士顿大学正在为其研究辩护。
BU 的国家新兴传染病实验室移除了一种 omicron 变异刺突蛋白，并将其附着在原始 COVID 毒株的病毒上。正如 Boston.com 报道的那样，这种融合病毒随后被发现在暴露于实验室小鼠时比天然存在的 omicron 变体更致命，而原始毒株则为 100%。
“携带 Omicron S 的病毒强烈逃避疫苗诱导的体液免疫，主要是由于受体结合基序的突变……而 Omicron 会导致轻度、非致命的感染，而携带 Omicron S 的病毒会导致严重的疾病并致死80%，”报告说。
该报告的结果尚未经过同行评审，其结论是，与其他变体相比，omicron 尖峰的突变允许该变体逃避任何已形成的免疫，并且不是 omicron 严重程度较低的原因。
一些头条新闻称这项研究是“玩火”，或声称该大学直接声称他们已经开发出一种致命的 COVID 毒株。波士顿大学首先在一份声明中澄清说，这项研究不是功能获得性研究，因此没有制造出更严重的 omicron 变体版本，而是降低了“病毒复制的危险性”。
阅读更多：马萨诸塞州获得 2500 万美元用于研究中心以抵御未来的流行病
“其次，该研究得到了机构生物安全委员会（IBC）的审查和批准，该委员会由科学家和当地社区成员组成，”声明继续说道。 “波士顿公共卫生委员会也批准了这项研究。此外，这项研究反映并强化了其他组织（包括 FDA）进行的其他类似研究的结果。最终，这项研究将通过带来更好的、有针对性的治疗干预措施来帮助对抗未来的流行病，从而提供公共利益。”
“他们夸大了这个信息，他们歪曲了这项研究及其整体目标，” BU Chobanian 和 Avedisian 医学院微生物学主席 Ronald Corley 在与波士顿大学研究新闻网站 The Brink 交谈时说。
据称，这项研究的存在还导致了 BU 研究人员与国家过敏和传染病研究所之间的裂痕。 NIAID 帮助资助了他们的工作，但 NIAID 微生物学和传染病部主任 Emily Erbelding 告诉 STAT，在拨款申请中没有具体提及他们的确切工作。
“我认为我们将在未来几天进行对话，”Erbelding 对 STAT 说。
据 The Brink 称，研究人员表示，这项研究是在实验室的 3 级生物安全设施中进行的，通过几个互锁的门进出他们的工作区。空间完全密封，实验室配备了适当、先进的过滤和净化技术。
“我们认真对待我们如何处理病原体的安全和保障，病毒不会离开正在研究它的实验室，”科利对 The Brink 说。 “我们的全部目标是为了公众的健康。这项研究是其中的一部分，它发现了病毒的哪一部分是导致严重疾病的原因。如果我们能理解这一点，我们就可以开发出开发更好疗法所需的工具。”
哪里说了武汉改造舟山蝙蝠了？
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
中共舟山蝙蝠病毒，E蛋白100%吻和，石正丽RATG13假病毒。你再怎么骂，这些还是事实。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
中共WHO说的，极不可能是人工制造，结果BU轻轻松松搞定。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
师傅说是就是，你们不明白是因为心不够诚，肚子的法轮转的不够。
师傅~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~~
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
你舌头上分子里的氢原子，跟武汉毒株 病毒里的氢原子，也是100%的吻合呢，全同粒子
☭主体思想の光辉万万年！
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
你们扯这些东西没用，如果有更硬的东西就拿出来，没有，我就不浪费时间了。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
在当时的环境下，土工确实是“极不可能是人工制造”啊。没错啊。
BU轻松搞定，代表了 美国、台湾和 土鳖的科技差距。
就像 台巴极不可能造出核武器搞地下核试验。
结果，北朝鲜能轻松搞定。这只能说明在核武器研发方面，台巴和北朝鲜存在技术差距。
☭主体思想の光辉万万年！
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
你杀人了。别扯那些没用的，你赶快把凶器交出来吧。（以子之矛攻子之盾，可乎？）
☭主体思想の光辉万万年！
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
比如，E蛋白就是指纹，基因。RATG13假病毒就是作伪证。
如果你也有就拿出来，我们看看。
如果你也有就拿出来，我们看看。
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
RATG13是真病毒，真实的存在，2013年就上传过序列了。
谁说RATG13是假病毒就让石正丽操他大爷！
有本事去生物杂志发文章说石正丽造假，该被剥夺院士头衔。在这里犬吠成什么体统！
☭主体思想の光辉万万年！
Re: 1450 ghobuster 说，我给了BU的链接了，可是我为什么从里面找不到武汉改舟山蝙蝠的内容呢？
五毛先把事实搞清楚再来搅混水吧，RATG13是石正丽在武汉病毒爆发后才发表的。